1. Andrew M, Carter C, O’Brodovich H, and Heigenhauser G. Increases in factor VIII complex and fibrinolytic activity are dependent on exercise intensity. J Appl Physiol. 1986; 60: 1917-1922.
2. Barron HV, Harr SD, Radfort MJ, Wang Y, and Krumholz HM. The association between white blood cell count and acute myocardial infarction mortality in patients > or =65 years of age: findings from the cooperative cardiovascular project. J Am Coll Cardiol. 2001; 38: 1654-1661.
3. Cassatella MA. The production of cytokines by PMN. Immunol Today. 1995; 16: 21-26.
4. Cusack MR, Marber MS, Lambiase PD, Bucknall CA, and Redwood SR. Systemic inflammation in unstable angina is the result of myocardial necrosis. J Am Coll Cardiol. 2002; 39: 1917-1923.
5. Distler JH, Pisetsky DS, Huber LC, Kalden JR, Gay S, and Distler O. Microparticles as regulators of inflammation. Novel players of cellular crosstalk in the rheumatic diseases. Arthritis Rheum. 2005; 52: 3337-3348.
6. El-Sayed M. Effects of exercise on blood coagulation, fibrinolysis and platelet aggregation. Sports Med. 1996; 22: 282-298.
7. El-Sayed M, Ali N, El-Sayed A. Aggregation and activation of blood platelets in exercise training. Sports Med. 2005; 35: 11-22.
8. El-Sayed M, El-Sayed Z, and Ahmadizad S. Exercise and training effects on blood haemostasis in health and disease: an update. Sport Med. 2004; 34: 181-200.
9. Enjeti AK, Lincz LF and Seldon M. Microparticles in health and disease. Semin Thromb Hemost. 2008; 34: 683-692.
10. Esmon CT, Fukudome K, Mather T, Bode W, Regan LM, Stearns-kurosawa DJ, and Kurosawa S. Inflammation, sepsis, and coagulation. Haematologica. 1999; 84: 254-259.
11. Faint RW, Mackie IJ and Machin SJ. Platelet aggregation is inhibited by a nitric oxide-like factor released from human neutrophils in vitro. Br J Haematol. 1991; 71: 539-545.
12. Faxon DP, Creager MA, Smith SC Jr, Pasternak RC, Olin JW, Bettmann MA, Criqui MH, Milani RV, Loscalzo J, Kaufman JA, Jones DW, and Pearce WH. Pearce atherosclerotic vascular disease conference: executive summary: atherosclerotic vascular disease conference proceeding for healthcare professionals from a special writing group of the American Heart Association. Circulation. 2004; 109: 2595-2604.
13. Gasser O and Schifferli JA. Activated polymorphonuclear neutrophils disseminate anti-inflammatory microparticles by ectocytosis. Blood. 2004; 104: 2543-2548.
14. Gillis S, Furie BC, and Furie B. Interactions of neutrophils and coagulation proteins. Semin Hematol. 1997; 34: 336-342.
15. Gorbet MB and Sefton MV. Biomaterial-associated thrombosis: roles of coagulation factors, complement, platelets and leukocytes. Biomaterials. 2004; 25: 5681-5703.
16. Goto S, Ikeda Y, Murata M, Handa M, Takahashi E, Yoshioka A, Fujimura Y, Fukuyama M, Handa S, and Ogawa S. Epinephrine augments von Willebrand factor-dependent shear-induced platelet aggregation. Circulation. 1997; 86: 1859-1863.
17. Hagberg IA, Roald HE and Lyberg T. Adhesion of leukocytes to growing arterial thrombosis. Thromb Haemost. 1998; 80: 852-858.
18. Huang ZC, Chien KL, Yang CY, Wang CH, Chang T, and Chen CJ. Peripheral differential leukocyte counts and subsequent mortality for all diseases, cancers, and cardiovascular diseases in Taiwanese. J Formos Med Assoc. 2003; 102: 775-781.
19. Ji LL. Antioxidants and oxidative stress in exercise. Proc Soc Exp Biol Med. 1999; 222: 283-292.
20. Ji LL, Katz A, Fu RG, Parchert M, and Spencer M. Alteration of blood glutathione status during exercise: The effect of carbohydrate supplementation. J Appl Physiol. 1993; 74: 788-792.
21. Lehmann M, Hasler K, Bergdolt E, and Keul J. Alpha-2-adrenoreceptor density on intact platelets and adrenaline- induced platelet aggregation in endurance-and nonendurance-trained subjects. Int. J. Sports Med. 1986; 7: 172-176.
22. Levi M, van der Poll T and Büller HR. Bidirectional Relation Between Inflammation and Coagulation. Circulation. 2004; 109: 2698-2704.
23. Li DB, Hua Q, Liu Z, Li J, Xu LQ, Wang S, and Jin WY. Association between inflammatory mediators and angiographic morphologic features indicating thrombus formation in patients with acute myocardial infarction. Chin Med J. 2009; 122(15): 1738-1742.
24. Libby P, and Aikawa M. Stabilization of atherosclerotic plaques: new mechanisms and clinical targets. Nat Med. 2002; 8: 1257–1262.
25. Mallat Z, Benamer H, Hugel B, Benessiano J, Steg PG, Freyssinet JM, and Tedgui A. Elevated levels of shed membrane microparticles with procoagulant potential in the peripheral circulating blood of patients with acute coronary syndromes. Circulation. 2000; 101: 841-843.
26. Mesri M and Altieri DC. Endothelial cell activation by leukocyte microparticles. J Immunol. 1998; 161: 4382-4387.
27. Mesri M and Altieri DC. Leukocyte microparticles stimulate endothelial cell cytokine release and tissue factor induction in a JNK1 signaling pathway. J Biol Chem. 1999; 274:23111-23118.
28. Mickelson JK, Lakkis NM, Villarreal-Levy Hughes BJ, and Smith CW. Leukocyte activation with platelet adhesion after coronary angioplasty: a mechanism for recurrent disease? J Am Coll Cardiol. 1996; 28: 345-353.
29. Moholdt TT, Amundsen BH., Rustad LA, Wahba A, Løvø KT, Gullikstad LR, Bye A, Skogvoll E, Wisløff U, and Slørdahl SA. Aerobic interval training versus continuous moderate exercise after coronary artery bypass surgery: a randomized study of cardiovascular effects and quality of life. Am Heart J. 2009; 158: 1031-1037.
30. Morel O, Toti F, Hugel B, Bakouboula B, Camoin-Jau L, Dignat-George F, and Freyssinet JM. Procoagulant microparticles. Disrupting the vascular homeostasis equation? Arterioscler Thromb Vasc Biol. 2006; 26: 2594-2604.
31. Munk PS, Butt N, and Larsen AI. High-intensity interval exercise training improves heart rate variability in patients following percutaneous coronary intervention for angina pectoris. Int J Cardiol. 2010; 145(2): 312-314.
32. Munk PS, Staal EM, Butt N, Isaksen K, and Larsen AI. High-intensity interval training may reduce in-stent restenosis following percutaneous coronary intervention with stent implantation: a randomized controlled trial evaluating the relationship to endothelial function and inflammation. Am Heart J. 2009; 158: 734-741.
33. Naruko T, Ueda M, Haze K, van der Wal AC, van der Loos CM, Itoh A, Komatsu R, Ikura Y, Ogami M, Shimada Y, Ehara S, Yoshiyama M, Takeuchi K, Yoshikawa J, and Becker AE. Neutrophil infiltration of culprit lesions in acute coronary syndromes. Circulation. 2002; 106: 2894-2900.
34. Nieman DC. Does Exercise Alter Immune Function and Respiratory Infections? President’s Council on Physical Fitness and Sports. 2001; 3: 1-8.
35. Perrin J, Lecompte T, Tournier A, Morlon L, Marchand-Arvier M, and Vigneron C. In vitro effects of human neutrophil cathepsin G on thrombin generation: Both acceleration and decreased potential. Thromb Haemost. 2010; 104: 514-522.
36. Piccin A, Murphy WG, and Smith OP. Circulating microparticles : pathophysiology and clinical implications. Blood Rreviews. 2007; 21: 157-171.
37. Pluskota E, Woody NM, Szpak D, Ballantyne CM, Soloviev DA, Simon DI, and Plow EF. Expression, activation, and function of integrinαMβ2 (Mac-1) on neutrophil-derived microparticles. Blood. 2008; 112: 2327-2335.
38. Preston RA, Jy W, Jimenez JJ, Mauro LM, Horstman LL, Valle M, Aime G, and Ahn YS. Effects of severe hypertension on endothelial and platelet microparticles. Hypertension. 2003; 41: 211-217.
39. Raghavan SAV and Dikshit M. Recent advances in the status and targets of antithrombotic agents. Drugs Fut. 2002; 27(7): 669.
40. Robertson JD, Maughan RJ, Duthie GG, and Morrice PC. Increased blood antioxidant systems of runners in response to training. Clin Sci. 1991; 80: 611-618.
41. Samis JA, Garrett M, Manuel RP, Nesheim ME, and Giles AR. Human neutrophil elastase activates human factor V but inactivates thrombin-activated human factor V. Blood. 1997; 90:1065-1074.
42. Steffel J, Luscher TF, and Tanner FC. Tissue factor in cardiovascular diseases-molecular mechanisms and clinical implications. Circulation. 2006; 113: 722-731.
43. Stief TW, Kurz J, Doss MO, and Fareed J. Singlet oxygen inactivates fibrinogen, factor V, factor VIII, factor X, and platelets aggregation of human blood. Thromb Res. 2000; 97: 473-480.
44. Suzuki T, Yamauchi K, Yamada Y, Furumichi T, Furui H, Tsuzuki J, Hayashi H, Sotobata I, and Saito H. Blood coagulability and fibrinolytic activity before and after physical training during the recovery phase of acute myocardial infarction. Clin Cardiol. 1992; 15: 358-364.
45. Thom SR, Yang M, Bhopale VM., Huang S, and Milovanova TN. Microparticles initiate decompression-induced neutrophil activation and subsequent vascular injuries. J Appl Physiol. 2011; 110: 340-351.
46. Van den Burg PJ, Hospers JEH, Mosterd WL, Bouma BN, and Huisveld IA. Aging physical conditioning, and exercise-induced changes in hemostatic factors and reaction products. J Appl Physiol. 2000; 88(5): 1558-1564.
47. Vischer U and Wollheim C. Epinephrine induces von Willebrand factor release from cultured endothelial cells: involvement of cyclic AMP-dependent signalling in exocytosis. Thromb. Haemost. 1997; 77: 1182-1188.
48. Wang JS. Intense exercise increases shear-induced platelet aggregation through enhancement of von Willebrand factor Binding ,glycoprotein IIb/IIIa activation and P-selectin expression. Eur. J. Appl. Physiol. 2004; 91: 741-747.
49. Wang JS, Cheng ML, Yen HC, Lou BS, and Liu HC. Vitamin E suppresses enhancement of factor VIII-dependent thrombin generation by systemic hypoxia. Stroke. 2009; 40(2): 656-659.
50. Wang JS, Chow S, Chen J, and Wong M. Effect of exercise training on oxidized LDL-mediated platelet function in rats. Thromb. Haemost. 2000; 83: 503-508.
51. Wang JS, Jen C, and Chen H. Effects of exercise training and deconditioning on platelet function in men. Arteriosc. Thromb. Vasc. Biol. 1995; 15: 1668-1674.
52. Wang JS, Li Y, and Chen J. Effects of exercise training and deconditioning on platelet Aggregation induced by alternating shear stress in men. Arteriosclero. Thromb. Vasc. Biol. 2005; 22: 454-460.
53. Wang JS, Lin C, Chen J, and Wong M. Role of chronic exercise in decreasing oxidized LDL-potentiated platelet activation by enhancing platelet-derived NO release and bioactivity in rats. Life Sci. 2000; 66: 1937-1948.
54. Warburton DER, McKenzie DC, Haykowsky MJ, Taylor A, Shoemaker P, Ignaszewski AP, and Chan SY. Effectiveness of high-intensity interval training for the rehabilitation of patients with coronary artery disease. Am J Cardiol. 2005; 95: 1080-1084.
55. Watts EJ. Haemostatic changes in long-distance runners and their relevance to the prevention of ischaemic heart disease. Blood Coagul Fibrinolysis. 1991; 2: 221-225.
56. Weiss C, Seitel G, and Bartsch P. Coagulation and fibrinolysis after moderate and very heavy exercise in healthy male subjects. Med. Sci. Sports Med. 1998; 30: 246-251.
57. Wilkie RP, Vissers MC, Dragunow M, and Hampton MB. A functional NADPH oxidase prevents caspase involvement in the clearance of phagocytic neutrophils. Infect Immun. 2007; 75: 3256-3263.
58. Wisløff U, Støylen A, Loennechen JP, Bruvold M, Rognmo Ø, Haram PM, Tjønna AE, Helgerud J, Slørdahl SA, Lee SJ, Videm V, Bye A, Smith GL, Najjar SM, Ellingsen Ø, and Skjærpe T. Superior cardiovascular effect of aerobic interval training versus moderate continuous training in heart failure patients-a randomized study. Circulation. 2007; 115: 3086-3094.
59. Zatta A, Prosdocimi M, Bazzoni G, Dejana E, and Del Maschio A. Inhibition of platelet function by polymorphonuclear leukocytes. J Lab Clin Med. 1990; 116: 651-660.
60. 張雅綸，＜低氧運動訓練對單核球釋放微粒調節凝血脢生成的影響 >，長庚大學，碩士論文，2010