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研究生:廖玉婉
研究生(外文):Liao, Yu-Wan
論文名稱:香蕉紅斑病病因之探討
論文名稱(外文):Etiology of banana red spot
指導教授:蘇鴻基蘇鴻基引用關係
指導教授(外文):Su, Hong-Ji
學位類別:碩士
校院名稱:國立臺灣大學
系所名稱:植物病蟲害學系
學門:農業科學學門
學類:植物保護學類
論文種類:學術論文
論文出版年:1993
畢業學年度:81
語文別:中文
論文頁數:60
中文關鍵詞:香蕉紅斑病組織培養苗基因突變球狀體
外文關鍵詞:banana red spotTC-seedingsgene defectspherial bodies
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菲律賓香蕉品種Umalag用組織培養苗在田間栽種結果,有 3﹪的植株會在
葉片、中肋及葉柄發生紅褐色斑點,係病因不明之新病害,稱之為香蕉紅
斑病。此病在台灣其他香蕉品系,如北蕉、仙人蕉、台蕉 1號及BF等不發
生。利用病組織分離、指示植物之病毒接種、石蠟切片、組織透化、徒手
切片、電顯切片等各種可能的病害診斷方法,檢驗病株的病因,皆沒有發
現真菌、細菌、擬菌質體、原生動物、高等寄生植物的存在,也沒有病毒
顆粒、病毒內涵體或病毒引起的病態細胞細胞質改變等各種病毒感沒所造
成的現象發生,即沒有發現傳染性致病因子存在。對健康苗所做之磨擦接
種、針刺接種、病組織心接種以及靠接接種等病毒接種方式,均無法傳染
本病至健株。病害在田間只有發生在組織培養苗上,分布零星,無向周圍
健株傳染蔓延的情形。此病只可經無性繁殖苗傳播,由罹病植株產生的吸
芽和病塊莖之組織培養苗一定會有紅斑病發生。因此推論此病可能為
Umalag品種在組織培養時,發生遺傳因子缺陷變異所造成之基因缺陷病。
光線促進此病發展,在弱光照下此病發生輕微。病株的導管中產生膠狀物
質充滿管中構成阻塞,葉肉細胞及其他薄壁細胞中產生異常代謝物,出現
多數大小不一之球狀物體被酚類親和性染劑染上,部分細胞發生胞質變質
而起凝集,終於壞死。
About 3% of banana seedlings derived from tissue culture(TC) of
Philippinose banana cultivar "Umalag" produced etiologically
unknown new disease so-called "banana red spot" inducing
redish brown spots on leaf blades, midribes and petioles. The
disease did not occur in other banana cultivars. Diagnostic
examination by means of tissue isolation, viral inoculation
of indicator plants, paraffin sectioning, tissue clearing, and
electronmicro- scopy revealed that no fungus, bacterium,
mycoplasms, virus or inclusions were found to be associated.
Inoculation of healthy banana seedlings with diseased
materials through mechanical, puncture, inarching, and
core inoculation methods failed to transmit the disease.
The disease occurred sporadically in the banana orchards
established with TC-seedling only, and it was not spread from
the diseased to the adjacent healthy plants. The disease was
transmitted only through vegetatively propagated seedlings,
i.e. suckers from diseased plants and TC-seedlings derived
from diseased rhizomes developed the disease. It was
assumed that the disease was caused by gene defect due to
muta- tion occurring during tissue-culture propagation of
Umalag cul- tivar. The xylem vessels of diseased plants
were generally plugged with gel matrix. Abnormal
metabolic products, and spherial bodies stained by phenolic-
affinitive dyes, were formed in the mesophyll cells and the
other parenchyma cells, some of which eventurely became
necrotic due to cytoplasm coagulation. The light affected
disease development; disease development was considerably
prohibited by low light intensity.
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