本實驗用大白鼠來試驗capsinolol是否能造成呼吸道發炎及探討其機轉。結果顯示
capsinolol所造成的血漿外洩程度約為capsaicin所誘發的50%左右，但不會產生隨濃度影
響漸增的效應。對組織細胞的影響引起環狀細胞的去顆粒反應，也較capsaicin低。以藥
物包裹左側頸迷走神經和上喉神經，七天後以capsaicin包裹的其左側次級支氣管用capsa
icin誘發神經性血漿外洩情形有顯著的減少，且以免疫染色法的處理發現物質P免疫活性
軸突的密度也顯著減少；這些結果與以capsaicin處理的相似，顯示capsaicin對組織的刺
激較弱，且可能和capsaicin相同經由感覺神經元的破壞，減少物質P的合成來降低或抑制
發炎反應。

Total proteins in corm apices of tuberose (Polianthes tuberosa L. cv. Double) were extracted from the different flower bud differentiation stages and were resolved using one-and two-dimensional sodium dodecyl sulfate-polyacrylamide gel electrophoresis. Many polypeptide bands in the flower development stage were more prominent in intensities than in the early floral initiation stage. Tuberose corms at three different stages were assayed for free and conjugate IAA. Free IAA in corm tissues increased 2.7-fold at the flower bud differentiation as compared to the vegetative stage. Corms from the early floral initiation stage contained less free IAA compared to the vegetative stage, whereas ester IAA and amide IAA increased dramatically. Moreover, the levels of free IAA increase and the levels of conjugate IAA decrease in the floral differentiation stage compared to the early floral initiation stage.